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While thealkylating compounds (or their metabolites) readily reactat more than 12 sites on DNA bases, the N7 position ofguanine and the N3 position of adenine are the most reac-tive DNA sites. So far, candidate “driving” mech-anisms in depression are envisioned largely in neuromod-ulatory and biochemical terms, including some form ofmonoamine deficiency (Schildkraut, 1965), cholinergicoveractivity (Janowsky et al., 1972), hypothalamic-pitu-itary-adrenal (HPA) stress axis alterations (Holsboer, 2000;de Kloet et al., 2005) that promote atrophic change in thehippocampus (Dranovsky and Hen, 2006), potential defi -cits in neuronal growth factors (Duman and Monteggia,2006), and associated alterations in corticotropin-releasingfactor (CRF), glucocorticoid receptor function, and brain-derived neurotrophic factor (BDNF)

So far, candidate “driving” mech-anisms in depression are envisioned largely in neuromod-ulatory and biochemical terms, including some form ofmonoamine deficiency (Schildkraut, 1965), cholinergicoveractivity (Janowsky et al., 1972), hypothalamic-pitu-itary-adrenal (HPA) stress axis alterations (Holsboer, 2000;de Kloet et al., 2005) that promote atrophic change in thehippocampus (Dranovsky and Hen, 2006), potential defi -cits in neuronal growth factors (Duman and Monteggia,2006), and associated alterations in corticotropin-releasingfactor (CRF), glucocorticoid receptor function, and brain-derived neurotrophic factor (BDNF). TheBezafibrate Infarction Prevention (BIP) registryhas also noted reduction in coronary events insubjects with high TG and low HDL-CH levels.Adverse effects and contraindications aresimilar to other fibrates.

(2002a)Dietary intake of antioxidants and risk of Alzheimer disease. For example, L-dopa(levodopa), the precursor of the neuromediators dopamine andnoradrenaline, easily crosses the blood-brain barrier

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